Font Size: a A A

Clinical Analysis Of30Cases Of Thalamic Infarction

Posted on:2013-02-13Degree:MasterType:Thesis
Country:ChinaCandidate:Y HuFull Text:PDF
GTID:2234330371485007Subject:Neurology
Abstract/Summary:PDF Full Text Request
ObjectiveTo explore the clinical features of different artery supple areas infarction of thala-mus,in further understand the characteristics of thalamic infarction.MethodsAccording to the literature,we divide thalamic infarction into four subtypes, anterolateral subtype (thalamotubercle artery supple area),posterolateral subtype (thalamogenilulate artery supple area),interior subtype (paramedian artery supple area),and dorsal subtype (posterior choroidal artery supple area).Retrospective analyses of the imageology characters and clinical features of30patients with thalamic infarction,to study according to the relationship between neuroanatomy and clinical features. ResultsIn the30cases of thalamic infarction, there are2anterolateral subtype cases,25posterolateral subtype cases,3interior subtype cases(all of them are bilateral interior thalamic infarction) and0dorsal subtype cases. Both of the2patients of anterolateral subtype have cognitive dysfunction,1patient is characterized by dysmnesia and acalculia, MMSE is19,1patient is characterized by aphasia. Among the25patients of posterolateral subtype,17patients have hemiparesis,16patients have hemisensory loss,2patients have dysarthria,1patient has thalamic pain,1patient has dystaxia. All the3patients of bilateral interior thalamic infarction have transitory disturbance consciousness and oculomotor paralysis,1patient has vertical gaze paralysis,1patient has cognitive dysfunction, mainly characterized by dysmnesia and acalculia.ConclusionDifferent arteries infartion involve different nuclei of thalamus. Thalamotubercle artery infarction mainly produce neuropsychological deficits; thalamo-genilulate artery infarction mainly produce contralateral hemisensory loss and hemiparesis,partly with pain syndromes;paramedian artery infarction mainly produce decreased arousal, dysmnesia, dementia, eye movement abnormalities and oculomotor paralysis; posterior choroidal artery infarction produce visual field loss, hemisensory loss and hemiparesis.
Keywords/Search Tags:thalamus, cerebral infarction, neuroanatomy, MRI
PDF Full Text Request
Related items