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Result: 81 - 97 | Page: 5 of 5
81.
A role for interleukin-17A as a mediator of sympathetic neuroanatomical remodelling during experimental colitis
82.
Matrix metalloproteinase activation and induction in keratinocytes by a purified thiol-proteinase from Porphyromonas gingivalis
83.
The Role Of High Mobility Group Box-1 Protein And Its Signaling Pathway In Pressure Overload-induced Cardiac Hypertrophy
84.
The Role Of Myeloid Differentiation 1 In Obesity-induced Left Ventricle Remodelling And Its Mechanism
85.
CaMKⅡ/cofilin/F-actin Singnaling Pathway Mediates Dendritic Spine Plasticity In Temporal Lobe Epilepsy Via Regulating The Neuronal Cytoskeleton Recombination
86.
Effects Of Astragaloside IV On Airway Inflammation And Airway Remodeling In Chronic Asthma Model Mice And Its Mechanism
87.
Study Of Pathogenesis Of Metastatic Bone Disease Based On Mathematical Model
88.
Effect Of Sox9 On The Regulation Of Atrial Fibrosis By TGF-β1
89.
Astragaloside IV Improves Pressure Overload-induced Myocardial Remodelling By Inhibiting The Activation Of NLRP3 Inflammasome
90.
Mechanism Of Matrix Reconstruction-Related Gene MXRA8 Affecting Malignant Phenotype Of Glioma
91.
The Mechanism Of KLF7/PFKL/ACADL Axis Regulating Cardiac Metabolic Remodelling During Cardiac Hypertrophy
92.
Construction Of Mouse Model Of Surgical Ventricular Restoration And Analysis Of Non-coding RNAs Changes In Reponse To The Surgery
93.
Mechanism Of Astragalus Yiqi Formula Regulates The Intestinal Flora/FXR Axis To Improve The Heart Failure
94.
The Therapeutic Effect Of Astragaloside Ⅳ On Peritoneal Membrane Remodelling By Inhibiting Peritoneal Mesothelial Cells NLRP3 Inflammasome Activation
95.
The Role And Mechanism Of High-Fat Stimulation Affecting NO Balance In Inducing Atrial Nerve Remodeling And Facilitating Atrial Fibrillation Via The CRIF1/eNOS/P21 Axis
96.
Study Of Galectin-3 And Sestrin2 And Their Correlation With Cardiac Remodeling In Patients With OSAHS Combined With Type 2 Diabetes Mellitus
97.
Phosphodiesterase 4 Regulates Intrinsic Cell Function And Participates In The Mechanism And Intervention Of Vascular Remodeling Disease
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