activated protein C - Globe Thesis

Keyword [activated protein C] Result: 1 - 20 \| Page: 1 of 2
1.	Study Of Etiology Of Cerebral Venous Thrombosis
2.	Development Of Activated Protein C Like Enzyme, A New Drug For Treating Severe Sepsis
3.	The Effects Of Activated Protein C On LPS-induced HUVEC
4.	The Effects Of Activated Protein C On Experimental Severe Acute Pancreatitis
5.	The Clinical Aspects And Molecule Machanisms Of A Hereditary Resistance To Activated Protein C Family
6.	Establishment Of Tissue-specific Thrombomodulin Overexpression Mouse Model
7.	Activated Protein C Inhibits Apoptosis In Podocytes
8.	Vein Thrombosis In Patients With Activated Protein C Resistance (apc-r) And The Anticoagulant Protein Defects
9.	Protective Effect Of Activated Protein C On Renal Injury Associated With Severe Acute Pancreatitis In Rats
10.	The Study On Activated Protein C Resistance And Factor V Leiden Mutation In Patients With Recurrent Spontaneous Abortion
11.	The Laboratory Study Of Activated Protein C And D-dimer In Deep Vein Thrombosis
12.	Study On The Relationship Of Mutation Of Prothrombin G20210A,Activated Protein C Resistance And Antithrombin In Patients With Coronary Heart Disease
13.	The Experimental Study On The Protective Effects Of Activated Protein C On Acute Gastric Mucosal Lesion
14.	Relationship Between The Structure And Anti-Thrombotic Mechanism Of Heparin-Derived Oligosaccharides
15.	Study On The Dependablity Between TM, APCR And Cerebral Infarction
16.	The Protein C System Of Bronchoalveolar Lavage Fluid In Bleomycin-induced Lung Fibrosis In Rat
17.	Effect Of Continuous Blood Purification On Prognosis And Endothelial Cell Function In Patients With Severe Sepsis
18.	The Relationship Analysis Between Activated Protein C (APC) And Urinary Microalbumin And Lower Extremity Vascular Disease In Patients With Type 2 Diabetes Mellitus
19.	The Dynamic Changes And Meanings Of Activated Protein C And Thrombomodulin In Severe Multiple Trauma Patients
20.	Study On Activated Protein C Inhibiting Endotheliocyte Apoptosis By Endoplasmic Reticulum Stress
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