Noxa - Globe Thesis - Globe Thesis

Keyword [Noxa] Result: 1 - 20 \| Page: 1 of 2
1.	Study Of The Correlation Between Cholesterol And Aβ Production
2.	The Experiment Study Of The Primary DNA Damage And The Expression Of Bax And Noxa In Mouse Cortical Neurons After Ischemia/Reperfusion Condition
3.	Cisplantin Induces Dual Modes Of Cell Death That Are Interrelated In Gastric Adenocarcinoma Cells
4.	Association Of BH3-only Proteins With Etoposide-Induced Apoptosis Of Gastric Adenocarcinoma Cells
5.	Effects Of Electroacupuncture On Mitochondria Apoptosis Pathway Of Pyramidal Cell In Hippocampus CA1 Area Of Rats With Vascular Dementia
6.	MRNA And Protein Expression Of Noxa In Lung Cancer
7.	Proliferation Inhibited And Apoptosis Induced Of Transfected Noxa Gene On Human Breast Cancer Cell Line MCF-7
8.	Synergistic Anti-tumor Activity Of ABT-737 And Celastrol
9.	The Blockage Of Autophagic Response Enhances The Anticancer Effect Of Neddylation Inhibitor MLN4924 In Liver Cancer
10.	B-AP15 Induces Cell Apoptosis Via Up-regulation Of Noxa In Esophageal Carcinoma Cells EC1 And Kyse 450
11.	Water Soluble PM2.5 Extracts Induces Apoptosis Via P53-Noxa-mediated ROS Generation In HBE Cells
12.	Upregulation Of NOXA By 10-hydroxycamptothecin Plays A Key Role In Inducing Fibroblasts Apoptosis And Reducing Epidural Fibrosis
13.	The Mechanisms Of Apoptosis Induction By Deubiquitylatinase Inhibitor P5091 In Esophageal Squamous Cell Carcinoma Cells
14.	The Mechanism Of Autophagy Regulation Of Apoptosis
15.	The Study Of Noxa And Puma Gene Induced Apoptosis Of Hepatocellular Carcinoma Cell Induced By HTERT Promoter
16.	Effect About Metformin Combined With Pemetrexed On Proliferation,apoptosis And Expression Of Mcl-1 And Noxa In Human Lung Adenocarcinoma A549 Cells
17.	The Mechanism Of USP18 Promoting The Apoptosis Of Liver Cancer Cells Induced By NDV
18.	ABT-263 Enhances The Effect And Mechanism Of Apigenin Against EGFR Mutation Tumors
19.	Structural studies of soluble guanylyl cyclase and its bacterial homologs
20.	Studies of the antitumor activity of alpha-TEA in human breast cancer cells
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