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Keyword [Microglia/macrophages]
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1. The Role Of MAPK Signaling Pathway On NO-mediated Secondary Injury In The Rat Spinal Cord
2. Studies On Class A Scavenger Receptor-mediated Cerebral Ischemia Injury
3. Aldose Reductase Regulates Microglia/macrophages Polarization To Influence Locomotion Recovery After Spinal Cord Injury In Mice
4. Aldose Reductase Regulates Microglia/Macrophages Polarization Through CREB After Spinal Cord Injury In Mouse
5. Peptide H11 Targeting Class A Scavenger Receptor Opposites Cerebral Ischemia/reperfusion Injury
6. Therapeutic Effects Of Diosgenin In Experimental Autoimmune Encephalomyelitis
7. Intravital Imaging Reveal Microglia Function In Melanoma Brain Metastasis And Neuropathic Pain
8. The Effects And Mechanism Of Necroptosis On The Polarization Of Local Macrophages/microglia Post Cerebral Ischemia In Mice
9. Study Of Therapeutic Effect And Underlying Mechanisms Of PI3K Inhibitor ZSTK474 In Mice With Cerebral Ischemia/Reperfusion Injury
10. Function Of M1 And M2 Microglia/Macrophages After Ischemic Stroke In Mice
11. Effects Of Lycium Barbarum Extracts On Microglia/Macrophages And Autophagy After Partial Optic Nerve Transection In Rats
12. Annexin A2 and microglia/macrophages in glioma progression
13. Human Adipose Mesenchymal Stem Cells Derived Exosomes Promote Recovery From Traumatic Brain Injury By Inhibiting Microglia/macrophages Activation In Rats
14. The Role Of Glioma Stem Cells In Inducing Infiltration Of Glioma Related Microglia/macrophages And Their Immunosuppressive Phenotype
15. Liraglutide Regulates Microglia/Macrophage Polarization By Activating The Nrf2/HO-1 Signaling Pathway And Its Protective Effect On Diabetic Rats With Cerebral Ischemia
16. Liraglutide Ameliorates Cerebral Ischemia Injury In Diabetic Rats By Up-regulating PPAR? To Regulate Microglia/macrophage Polarization
17. The Correlation Between The Change Of Microglia/Macrophage Polarization Type And Tumor Biological Characteristics In Recurrent Glioma
18. Bushen Yizhi Anti-Aging Prescription Improve Cognitive Function In APP/PS1 Mice Via The Inhibition Of TLR4/MyD88/NF-?B And The Polarization Of M2
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