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Keyword [HPV16 E6/E7]
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1. Studies On The Expression And Immunogenicity Of The Modified Human Papillomavirus Type 16 E6/E7 Fusion Protein As Well As Its Antitumor Effacacy
2. The Establishment Of Lentiviral-vector With ShRNA Targeting Common Promoter Of HPV16E6and E7Oncogenes And Inhibition Of Cervical Cancer Cell Growth In Vitro And In Vivo
3. Construction Of SiRNA Expressing Vector Systerm Of Inhibiting HPV16 E6/E7 Activity
4. Significance Of P16INK4a And P14ARF Expression In Cervical Carcinogenesis
5. Expression, Purification, And Serum Antibody Detection Of HPV16 E6, E7
6. Effect Of HPV 16 E5 On E6 And E7 Gene Induced Human Oral Immortalized Epithelial Cell
7. In-depth Study Of Chemically Synthesized Sirna Silence Siha Cell E6 Gene Expression
8. Analysis Of The Relationship Brtween Multiple HPV Infection And HPV16 E6/E7 Genetic Variation With The Development Of Cervical Cancer
9. Effects Of HPV16 E6/E7 Transcriptional Gene Stable Silencing And CyclinD1 Overexpressi-on Respective On Proliferation Apoptosis And Cyclical Factors Of Human Cervical Cancer SiHa Cells
10. Effects Of 5-aza-2 '-deoxycytidine On The Expression Of HPV16 E6/E7,P53,pRb And Twist1 In Squamous Cervical Cancer SiHa Cells In Vitro
11. HPV16 E6/E7 Upregulated The MRNA Expression And The Amplification Of HTERC By Releasing LKB1 To Promote Phosphorylation Of SP1 In Lung Cancer Cells
12. HLA Restrictive HPV16 E6,E7 Antigen Specific T Cell Immune Response And Clinical Characteristics And Prognosis Of Cervical Squamous Cell Carcinoma
13. The Mechanisms Of HPV16 E6/E7 Inducing Primary Keratinocyte Malignant Transformation By DNA Methylation Pathway
14. HPV16 E6/E7 Up-regulate The Expression Of Both HIF-1? And GLUT1 By Inhibition Of RRAD And Activation Of NF-?B In Lung Cancer Cells
15. HPV16 E6/E7 Promote The Glucose Uptake Of GLUT1 In Lung Cancer Cells Through Downregulation Of Txnip Due To Inhibition Of PTEN Phosphorylation
16. HPV16 E6 And E7 Lead To The Increase Of GLUT1 Transport Activity In Cervical Cancer Cells By Promoting The Carcinogenic Activity Of P65 Suppressing The Nuclear Output Of HIF-1?
17. HPV16 E6/E7 Inhibited The Antitumor Activity Of LKB1 By Down-regulating KIF7 And Activation Of MiR-106a In Lung Cancer Cells
18. HPV16 E6/E7 Promotes GLUT1 Plasma Membrane Translocation And Glucose Uptake By Inhibiting NBR2 Phosphorylation Of AMPK In Lung Cancer Cells
19. HPV16 E6/E7 Promote The Translocation And Glucose Uptake Of GLUT1 By PI3K/AKT Pathway Via Relieving MiR-451 Inhibitory Effect On CAB39 In Lung Cancer Cells
20. The Mechanism Of HPV Promoting Invasion And Metastasis Of Cervical Cancer Cells Through Upregulating ICAT
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