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Keyword [EVI1]
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1. Establishment And Characterization Of Two Human Acute Myelomonocutic Leukemia Cell Lines, JIH-3 And JIH-6 With Del(7p) And T(9;22), Respectively
2. Critical Role Of MiR-9in Myelopoiesis And Evi1-induced Leukemogenesis
3. The Clinical Study And Mechanistic Exploration Of BCL11A In Myeloid Leukemia
4. Suppression Of Evi1 Gene Alleviates Osteoporosis Through Promoting Differentiation Of BMSCs To Osteoblasts
5. Clinical Significance And Regulatory Mechanisms Of EVI1 Overexpression In Acute Myeloid Leukemia
6. Prognostic Significance Of EVI1 Expression In Adult Acute Myeloid Leukemia Patients
7. Expression And Clinical Significance Of EVI1 And BRE Gene In MLL-rearranged Acute Leukemia
8. Expression Of EVI1 And Its Clinical Significances In Acute Myeloid Leukemia And Acute Lymphoblastic Leukemia
9. Clinicalcharacteristics And Prognostic Analyze Of Acutemyeloid Leukemia Cases With EVI1 Gene Positive Expression
10. Evi1 Promotes Proliferation Of Nasopharyngeal Carcinoma Via MiRNA-33 Mediated Downregulation Of P53 Expression
11. MicroRNA-22 Reduces Neointima Formation Via Regulating Smooth Muscle Cell Phenotypic Modulation
12. A Preliminary Study Of The Pathogenesis Of EVI1 Gene In Children Acute Lymphoblastic Leukemia
13. Studies On The Expression And Prognostic Significance Of The Gene EVI1 In Acute Lymphoblastic Leukemia (ALL)
14. The Role Of Evi1/Smad3 In Committed Differentiation Of Leukemia Cells Into Monocytes/Macrophages
15. Preliminary Study About The Impact Of Evi1 Expression On Biological Behavior And Chemotherapy Sensitivity In Ovarian Cancer Cells
16. EVI1 Promotes Cell Proliferation And Suppresses Apoptosis In Hilar Cholangiocarcinoma Via PTEN/AKT Signalling Pathway
17. Expression Of EVI1 Gene And Its Clinical Significance In Pediatric Acute Myeloid Leukemia
18. The Regulation Of EVI1 Gene For Hematopoietic Transcription Factors And Evaluation Of Arsenic Trioxide
19. The Outcome Of AML Patients With MLL Gene Rearrangement Undergoing Allogeneic Hematopoietic Stem Cell Transplantation
20. Identifying a Potential Molecular Therapeutic Target for MLL-AF9 Leukemi
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