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Keyword [C2C12 myoblasts]
Result: 1 - 20 | Page: 1 of 2
1. Rehabilitation Of Red Light From Light Emitting Diode Array On Ethanol Inhibited C2C12 Myoblast Proliferation
2. Effect Of Different Concentrations Of ALLN On Proliferation And Apoptosis Of C2C12 Myoblasts
3. Effects Of Alcohol On Expression Of E3 Ligase Atrogin-1 In C2C12 Myoblasts
4. Effects Of Calpastatin Peptide And Calpain Inhibitor ? On Metabolism Of C2C12 Myoblasts In Tumor Microenvironment
5. The Function And Mechanism Of Liraglutide On The Differentiation Of C2C12 Myoblasts And The Expression Of FNDC5 In Myotubes
6. Activation Of MTOR Pathway Is Involved In Lipid Disorder Under Condition Of Hypoxic Environment
7. Atractylenolide-1 Attenuates TNF-?-induced Apoptosis And Autophaghy In Mouse C2C12 Myoblasts
8. Study Of Hypoglycemic Activity And The Mechanism Of Extracts From Edgeworthia Gardneri(Wall.) Meissn
9. Adiponectin Modulates Oxidative Stress-induced Mitophagy And Protects C2C12 Myoblasts Against Apoptosis
10. Effects Of Calpastatin Peptide,Calpain Inhibitor ? And Calpeptin On Expression Of AP-1 And PARP Of C2C12 Myoblasts In Tumor Microenvironment
11. Effect Of Calpastatin Peptide?Calpain Inhibitor ??Calpeptin On Proliferation And Apoptosis Of C2C12 Myoblasts In Tumor Microenviroment
12. Clinical And Basic Research On Adipose Tissue, Muscle Tissue And Its Factors And Metabolic Syndrome
13. Low Frequency Pulsed Electromagnetic Field Promotes C2C12 Myoblasts Proliferation Via Activation Of MAPK/ERK Pathway
14. Alcohol Acts Via P38 MAPK To Induce Expression Of Atrogin-1 In The Differentiation Process Of C2C12 Myoblasts
15. Lead Acetate Exposure Induced Oxidative Stress And Apoptosis In C2C12 Myoblasts
16. NMR-based Metabolomic Analysis Of The Effects Of Alanyl-glutamine Supplementation On C2C12 Myoblasts Injured By Energy Deprivation
17. Rab8a Regulates The GLUT4 Translocation In C2C12 Myoblasts
18. Study On Orotective Effect And Mechanism Of Angelica Sinensis Polysaccharide On TNF-?-induced Injury In Mouse C2C12 Myoblasts
19. mTORC1 activation is reduced in an in-vitro model of Pompe disease
20. Transcriptional control of metabolism and the response to ischemia in muscle
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