| At present,the emotion regulation disorder of depression has become a new hot spot in the study of emotion disorder.There are two different theoretical hypotheses in this field.Emotion regulation strategy preferences arise because of their imperfect social cognitive functioning,which leads to deactivation between their emotion regulation strategy choices;and secondly,patients’ repeated failure to use a certain strategy leads to their choice rejection of that strategy.Therefore,in this paper,we selected a subclinical risk sample for depression using a follow-up questionnaire to examine the flexibility of their choice of emotion regulation strategies in daily social interactions.In addition ERP instruments were used to examine the functional performance of the subclinical sample on cognitive reappraisal strategies in order to understand the presence of functional impairment.The results of study 1 found that subclinical individuals used less cognitive reappraisal strategy,more attention transfer strategy and showed some emotional strategy inactivation than healthy individuals.We believe that this is indicative of impaired emotion regulation in subclinical individuals,and since our questionnaire examines their ability to regulate emotions in daily life,we believe that the results indicate a degree of impaired social cognition and identify the type of emotion regulation strategies that are rejected by this group of individuals.The results of study 2 found that there were significant differences in N2 components before and after cognitive reassessment,but no differences in P3 components.Subclinical individuals are not affected by depression in their regulation of emotional intensity and try to moderate the effects of negative stimuli.However,they are unable to adjust their attention to negative stimuli through cognitive reappraisal strategies,which is the hallmark of their "subclinical" nature.Overall our results validate both orientations and lead to the prospective conclusion that emotion regulation impairments are not only found in clinical samples,but also in subclinical individuals at risk. |