Receptor interacting protein - Globe Thesis

Keyword [Receptor interacting protein] Result: 41 - 60 \| Page: 3 of 4
41.	Expression Of RIP140 In Human Hepatocellular Carcinoma And Its Clinical Significance
42.	Expression Of Receptor-interacting Protein Kinase 3 (RIP3) In Rat Neuropathic Pain Model
43.	The Protective Effect Of Necrostatin-1 On Sepsis Associated Acute Kidney Injury
44.	The Experimental Study Of Androgen Receptor Interacting Protein In Male Mononuclear Cells
45.	The Role Of Activin Receptor-interacting Protein 1 And Activin A In Traumatic Brain Injury
46.	The Role Of Activin Receptor-interacting Protein 2 In Traumatic Brain Injury
47.	Receptor-interacting Protein 1 Is A Key Mediator In Oxidative Stress-induced Necroptosis Of Melanocytes
48.	Experimentall Study On Necroptosis Of Central Nernervous System Induced By Manganese Exposure In Rats
49.	The Role Of RIPK1 Kinase Activity In The Pathogenesis Of Perioperative Stroke
50.	The Role Of Stat3/NF-κB In The Development And Drug Resistance Of MLL-r Acute Myeloid Leukemia
51.	TRIP6 Regulates The Proliferation,Migration,Invasion And Apoptosis Of Osteosarcoma Cells By Activating The NF-κB Signaling Pathway
52.	Study On The Role And Mechanism Of RIP3 In Macrophages In Liver Fibrosis
53.	The Role Of RIP1 Mediated Necroptosis Insteatotic Liver Ischemia Reperfusion Injury
54.	Mechanism And Protection Of Programmed Necrosis Induced By Sodium Salicylate In Rat Spiral Ganglion
55.	Expression And Significance Of Necroptotic Proteins In Vitreous After Rhegmatogenous Retinal Detachment
56.	Discussion On The Mechanism Of Yishen Juanbi Pill Regulating The Differentiation Of Osteoclasts Based On EphrinB2 Signal Passway
57.	The Role And Mechanism Of Renal Tubular Epithelial Cells RIPK3 In The Progression From Acute Kidney Injury To Chronic Kidney Disease
58.	Role And Molecular Mechanisms Of Artesunate In Programmed Eosinophilic Death In Asthma
59.	Mechanism Of MiR-200c-3p/RIP2/NF-κB Regulation Of LPS-induced Activation Of BV2 Microglia
60.	The Role Of RIP3/CaMKⅡ/Pyk2 Pathway In Programmed Cell Death In Cerebral Infarction
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